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Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

Neoplasms hide in hair follicles to avoid the immune system.

Keratin 6A increases skin inflammation, suggesting it could be a target for treating certain skin diseases.

Allergic contact dermatitis may promote hair growth by activating hair follicle stem cells.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

CGRP-IR axons may help maintain and renew tissues.

The method increases stem-like cells for better skin regeneration.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

CD34+ and CD34- melanocyte stem cells have different regenerative abilities.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Severe CCCA may be biologically and clinically different from milder forms.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Inflammation helps stem cells repair tissue by directing their behavior.

Candida auris uses the immune response to colonize hair follicles.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

p120-catenin helps control skin inflammation by regulating cadherin levels.

Targeting cholesterol, fatty acids, fibrosis, and mast cells may help treat CCCA.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

CD34 is absent in most basal cell carcinoma cells but present in surrounding skin.

Alopecia areata involves immune system changes, especially in severe cases, with potential new treatment targets identified.

Higher CRBP1 levels are linked to more severe alopecia areata.

Basonuclin 1 (BNC1) helps skin cells multiply and move, which is crucial for wound healing.