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The calcineurin/NFAT pathway plays a significant role in the development and growth of a type of skin cancer called cutaneous squamous cell carcinoma.

VDC-1101 shows potential as a treatment for canine cutaneous T-cell lymphoma.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Blood-derived CD34+ cells speed up healing, reduce scarring, and regrow hair in skin wounds.

Wnt and Notch pathways are crucial for repairing blood stem cells after damage.

Skin cells can naturally limit the growth of cancerous changes by balancing cell renewal and differentiation.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Different types of resting melanocyte stem cells have unique characteristics and vary in their potential to become other cells.

Stat3 affects skin cancer development by altering keratinocyte stem cell behavior.

CS12192 effectively treats alopecia areata with better safety than current options.

Ritlecitinib may help treat alopecia areata by protecting hair follicles.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

LGR5 and LGR6 are expressed differently in various skin tumors, which may offer clues about their origins.

EBV infection increases a specific keratin variant in carcinoma cells, possibly affecting cell structure and cancer progression.

CCCA may be caused by both hair traction and an immune response.

CCL5 is important for the hair growth potential of human dermal papilla cells.

Langerhans' cells are not involved in hair depigmentation in these mice.

Hair follicle cells resist turning into skin cells.

CD8+ T cells re-expressing CD45RA may predict treatment resistance in severe alopecia areata.

Stat3 activation increases hair follicle progenitors but reduces bulge region stem cells.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Adipose-derived stem cell-conditioned medium can reduce melanoma cell growth and spread.

Chemotherapy-induced hair loss is partly due to decreased laminin-511 and increased laminin-332.