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Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Blocking miR-27a increases sheep hair follicle stem cell growth and decreases cell death, which could help improve wool quality and treat hair loss.

TCDD changes skin cell growth and keratin production in mice.

A new test helps find drugs to treat head and neck cancer by targeting c-Rel.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

LL-37 helps stem cells grow and move, aiding tissue regeneration and hair growth.

CD1d expression in scalp skin and hair follicles changes with the hair cycle and may help protect against microbes.

B cells can both help and hinder the body's defense against melanoma.

CD44 variant changes start alopecia areata, but don't maintain it.

Blocking Rab27a slows hair growth, while blocking Rab27b encourages it.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

REV7 is crucial for genome stability and cancer treatment, making it a potential target for therapy.

Blocking certain immune signals can reduce skin damage from radiation therapy.

p21waf1/cip1 and p27kip1 help in hair follicle differentiation in rats.

CD133+ cells are crucial for hair growth.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

JAM-A helps hair regrowth in alopecia areata by protecting VCAN in skin cells.

BerEP4 and CD34 staining can help tell apart tricholemmoma from basal cell carcinoma.

Versican in dermal papilla cells is crucial for healthy hair growth.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Diphencyprone treatment increases CD8 lymphocytes in the scalp, which is associated with hair regrowth in alopecia areata patients.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

CXCR2 in skin cells promotes tumor growth.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

CD26+ fibroblasts improve skin healing and integration better than CD26− fibroblasts.

CEP135 may predict cancer outcomes, and targeting PLK1 could help treat certain sarcomas.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.