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The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.

Sex and race affect immune responses and treatment outcomes in Hidradenitis suppurativa.

Mohs micrographic surgery is effective for treating basal cell carcinoma on the eyelid, while radiotherapy has higher recurrence rates, and topical vitamin D3 may reduce and delay BCC formation in mice.

Researchers found a way to make rat hair follicle cells start turning into motor neuron-like cells, but couldn't fully turn them into working motor neurons.

The gp130 receptor helps in tissue regeneration and disease progression, and manipulating it could improve healing and prevent disease.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Some skin growths with mucin can form hair follicles and resemble skin cancer, but a special stain can help tell them apart.

Platelet-rich plasma doesn't prevent hair loss from chemotherapy, and mononuclear cells only help against hair loss from one specific chemotherapy drug.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Targeting vitamin D and androgen receptors may effectively treat triple-negative breast cancer, especially with chemotherapy.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

Hair follicle cells resist turning into skin cells.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

New hair regrowth model introduced, imiquimod kills skin cancer cells, T-cadherin loss makes skin cancer more invasive, no strong link between PTCH1 gene and skin cancer after transplant, and male teens more likely to have hereditary hair loss.

Checkpoint inhibitor therapy can cause skin issues, from mild rashes to severe reactions.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Bipotent dermal stem cells and specific macrophages are crucial for hair regrowth and regeneration.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

Versican in dermal papilla cells is crucial for healthy hair growth.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

CCCA may be a fibroproliferative disorder, and anti-fibrotic therapies could help.

Four specific genes are linked to keloid formation and could be potential treatment targets.