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Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

Tofacitinib and low-dose IL-2 may help maintain hair regrowth in alopecia areata without ongoing treatment.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Bone marrow-derived cells can lead to skin inflammation and tumors in mice.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

Disrupting Bcl-xL in mice reduces skin cancer risk.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

Arg1+ macrophages may play a role in causing alopecia areata.

A rare type of skin lymphoma was identified, affecting hair follicles and sweat glands.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

CK 19 expression is higher in more severe skin cancers.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

CCCA may be caused by both hair traction and an immune response.

T cell subsets are crucial in kidney cancer, and a new model predicts patient outcomes using key genes.

circCFAP20DC helps goat ovarian cells grow, aiding follicle development.

A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.

Kaposi's sarcoma lesions might originate from benign tissue changes.

A man developed facial skin lesions after a stem cell transplant, which improved with specific treatments.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Nonimmunogenic forms of keratins K71 and K31 can delay and prevent alopecia areata.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.