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ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Soluble CD83 speeds up wound healing and reduces scarring.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

LRIG1 is linked to better survival in Merkel cell carcinoma.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

Removing certain hair follicle stem cells worsens skin reactions to allergens.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

Abnormal Hedgehog signaling in blood cancers may help tumors grow and resist chemotherapy, suggesting potential for targeted treatments.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Keratin 75 might be important in oral cancer progression.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

γδ T cells adapt uniquely to different tissues in mice.

CD200 is not a reliable marker for identifying stem cells in all skin types.

Alopecia areata patients have fewer protective regulatory B cells, which may contribute to the disease.

Activating Notch signaling can kill basal cell carcinoma cells.

Clonally expanded CD8+ T cells cause alopecia areata.

CD34 is absent in most basal cell carcinoma cells but present in surrounding skin.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

Targeting specific T cells may help treat alopecia areata.

ABCG2 protein marks stem-like skin cells in human epidermis.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

CD34 is crucial for skin tumor development in mice.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

CD28 is a promising target for treating alopecia areata with belatacept.

CD28 is a promising target for treating alopecia areata with belatacept.