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Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

A form of vitamin E promotes hair growth by activating a specific skin pathway.

ILK is essential for skin development, pigmentation, and healing.

Desmosomes are crucial for skin and heart development, and JNK may help regulate them.

Dauricine may help treat certain brain cancers by reducing cancer cell growth and promoting cell death.

A pathway helps maintain long telomeres in both stem and cancer cells.

KY19382 helps regrow hair and create new hair follicles.

MPA increases cancer spread by boosting Eph A2 activity.

Researchers created immortal human skin cells with constant testosterone receptor activity to study hair loss and test treatments.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.

Inhibiting ODC can prevent UV-induced skin cancer.

K15 and Id3 are important in hair follicle regeneration, with K15 increasing in early stages and Id3 responding later.

Id2 gene helps keep hair follicle stem cells inactive.

AP-1 controls tumor cell type by affecting key signaling pathways.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Keratins K6 and K16 are expressed more freely in regenerating mouse skin than K1 and K10.

Pygo2 is important for early growth and progression of intestinal tumors, and could be a target for treating cancers with certain mutations.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

CYLD mutations cause a variety of skin tumors with symptoms starting around age 16, and treatments are currently limited.

The PML protein helps prevent skin cancer in mice.

Basal cell carcinomas and squamous cell carcinomas have different gene activity patterns, suggesting unique treatment approaches.

Mutations in the HOXC13 gene cause hair and nail development issues.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Keratoacanthoma changes as it grows, showing different hair follicle characteristics at each stage.

JAK/STAT1 pathway causes hair loss during chemotherapy by reducing Shh in hair follicles.

A KRT32 gene variant causes loose anagen hair syndrome.