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A protein from fat-derived stem cells, DKK1, is linked to hair loss and blocking it may help treat alopecia areata.

New cancer treatments show promise in reducing tumor growth and improving skin regeneration in mice.

Keratins 6, 16, and 17 increase in damaged or diseased skin and may help diagnose skin issues.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Reducing SOX2 in colorectal cancer cells decreases tumor growth and self-renewal.

EGFR helps control how hair grows and forms without needing p53 protein.

A new gene mutation may allow some piebaldism patients to regain skin color in white patches.

A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

S100A6 protein is linked to disease progression, especially in cancers.

Activating Kras in mouse skin causes excess skin and hair loss.

Ritlecitinib may help treat alopecia areata by protecting hair follicles.

A new one-step test can quickly identify skin cancer during surgery.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

ISCK03 stops melanin production in human melanoma cells and lightens skin color in mice and guinea pigs.

Cyclosporin A promotes hair cell growth and affects protein kinase C levels.

A new gene location for Keratosis follicularis squamosa was found on chromosome 7p14.3-7p12.1.

Mice with human skin protein K8 had more skin problems and cancer.

Two new gene clusters important for hair formation were found on human chromosome 11.

Human basal cell carcinomas and squamous cell carcinomas have unique gene expression patterns not fully mirrored in mouse models.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Some Greek melanoma patients have gene mutations linked to increased cancer risk, a new color feature helps diagnose melanoma, the incidence of a skin condition in the Netherlands is rare, and a gene possibly affects male-pattern baldness.