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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Ginsenosides Rb₁ and Rd may help prevent hair loss by promoting hair cell growth.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Mutations in hKAP1 genes may cause hereditary hair disorders.

Decorin helps hair cells grow and move, and keeps hair growth phase going in mice.

Inactive hair follicle stem cells help prevent skin cancer.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

A rabbit gene important for hair development was identified and detailed.

ILC1-like cells can cause alopecia areata by themselves.

Ginkgolide B and bilobalide help mink hair grow by making skin cells healthier and boosting a key growth protein.

TP0427736 may help treat hair loss by blocking a specific protein and promoting hair growth.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

Breast cancer treatments work better with AR activation, improving results and reducing side effects.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Fluphenazine and iloprost can induce hair growth.

Inhibiting Bmp signaling disrupts hair growth and differentiation.

PRC2 is not essential for hair follicle stem cell maintenance or hair growth.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Gefitinib can cause hair loss without scarring.

KAP genes are crucial for hair development and show both shared and unique traits in humans, chimpanzees, and baboons.

New hair regrowth model introduced, imiquimod kills skin cancer cells, T-cadherin loss makes skin cancer more invasive, no strong link between PTCH1 gene and skin cancer after transplant, and male teens more likely to have hereditary hair loss.

A specific gene mutation causes severe skin and nail issues and hair loss.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

New medicines that block a specific pathway are showing promise for treating severe hair loss but need more testing for safety and effectiveness.