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A special receptor in sensory nerve endings helps control how they respond to stretching.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.

Higher PHGDH levels cause unusual melanin buildup in hair follicles.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

ME1 and PPAR signaling may influence hair loss in androgenetic alopecia.

Mrp3 helps in wound healing and hair growth.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Blocking mTORC1 reduces skin tumor growth in mice.

Disrupting a specific protein's function in hair follicle stem cells triggers their activation and a self-healing process.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Multi-peptide factors from fibroblasts may stimulate hair growth by increasing growth factors and β-catenin in hair cells.

The study showed that a specific DNA sequence can control gene expression in hair growth areas of mice.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.

Blocking mTORC1 activity with rapamycin could help increase hair pigmentation and growth, potentially reversing gray hair.

Stat3 activation increases hair follicle progenitors but reduces bulge region stem cells.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

Activating TLR3 boosts autophagy gene expression in skin cells.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

EGFR hyperactivation increases sebaceous gland size and sebum production in mice.

A protein in plants needs to bind two lipids to help with root hair growth, and this process is similar across different plant species.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

A specific gene mutation is linked to a hereditary form of dystonia that responds well to certain medications.

Activating TRPV1 can boost hair growth by involving neurons, macrophages, and fibroblasts.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

Allergic contact dermatitis may promote hair growth by activating hair follicle stem cells.

PGA-4HGF may help treat hair loss by activating hair growth pathways and extending the hair growth phase.