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Vitamin D3 and its analogues affect skin cell growth and development depending on cell density, calcium levels, and serum presence.

Dihydrotestosterone increases the activity of an enzyme in pubic skin cells that converts testosterone to dihydrotestosterone.

Progesterone byproduct 5αP stimulates mammary tumor growth, but finasteride can suppress it.

A protein called CBP is found in prostate cancer and can increase the effectiveness of certain prostate cancer treatments.

Saw palmetto supplements may reduce prostate cancer cell growth without being toxic.

A yeast-based test can detect the steroid methyltestosterone in urine longer than traditional methods.

Some newly made compounds are promising for treating enlarged prostate, hair loss, viruses, and prostate cancer, and might be better than current drugs.

Scientists have developed a new method to detect steroid abuse in athletes using cell-based tests, which could be the future of anti-doping methods.

Steroid-producing capabilities in certain cancers may contribute to treatment resistance.

Estrogen affects scalp cell types differently, enhancing some wound healing processes.

The vitamin D receptor can work without its usual activating molecule.

17β-Estradiol slows wound healing in male mice through estrogen receptor-α.

GnRH treatment can reduce breast cancer cell invasion.

Adenosine complex helps increase hair thickness and density in hair loss.

Dutasteride and finasteride affect different cell types differently.

Inhibiting 5α-reductase increases progesterone levels in late pregnant mares.

Germacrone from Curcuma aeruginosa may help treat conditions related to male hormones by blocking a specific enzyme.

Blocking glucocorticoid receptors improves glucose metabolism in a PCOS mouse model.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

Cells from balding scalps have more androgen receptors than cells from non-balding scalps.

The study found that certain microRNAs are higher in the cells and lower in the fluid of women with a specific type of polycystic ovary syndrome, and one microRNA could potentially help diagnose the condition.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

Finasteride may improve prostate cancer treatment outcomes.

Dihydrotestosterone causes p27 protein breakdown in ovarian and breast cancer cells by binding with SKP2.

Dihydrotestosterone increases growth and spread of human brain cancer cells, and blocking its formation might help treat this cancer.

Increased PPARGC1α relates to hair thinning in common baldness.

PF-05314882 selectively activates androgen receptors without much effect on prostate and may help in prostate cancer treatment and hair loss prevention.

Vitamin D may help regulate cholesterol and influence prostate cancer development.

Extracellular vesicles and androgen receptors may help identify prostate cancer resistance and reduce SARS-CoV-2 infection.

RU58841, a nonsteroidal anti-androgen, showed potential as a topical treatment for hair loss, increasing hair density, thickness, and length without systemic side effects in Stumptailed Macaques.