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PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

FOXC1 is essential for keeping hair follicle stem cells inactive and maintaining their environment for healthy hair growth.

The human K5 promoter controls specific gene expression in skin cells, with key regulatory elements near the TATA box.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

HDAC6 helps keep ovarian follicles dormant, extending female fertility.

The trichohyalin gene is located at chromosomal region 1q21 with other skin-related protein genes.

Keratinocyte gene expression is controlled by multiple modules with specific binding sites.

The K5 promoter controls gene expression in skin cells, with specific DNA segments crucial for targeting and regulation.

Proper tissue repair in adult skin requires specific histone hypomethylation.

DNA methylation controls hair follicle gene expression in cashmere goats.

CIP/KIP proteins help stop cell division and support hair growth.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

Keratin gene regulation is similar across mammals, affecting hair follicle differentiation.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Silencing certain circadian clock genes increases skin pigmentation.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

DNA methylation likely doesn't cause different lambskin patterns in Hu sheep.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Increasing mir-302 turns human hair cells into stem cells by changing gene regulation and demethylation.

HT-scCAT-seq helps understand gene regulation in embryonic skin development.

HDAC1 is crucial for skin development and preventing tumors.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Mice without certain skin proteins had abnormal skin and hair development.

MOF controls skin development by regulating genes for mitochondria and cilia.