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The N363S gene variant does not cause higher adrenal androgen levels in women with polycystic ovary syndrome.

Mechanosensory neurons adapt to different skin types after birth.

Scalp hair follicle cells help protect and heal skin in certain skin conditions.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Higher CRHR1 levels in AA patients lead to increased inflammation.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Lower SMAD2/3 activation predicts more severe skin cancer.

TGF-β signaling is essential for hair follicle regeneration during wound healing.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

A specific gene mutation causes a severe skin disorder in a family.

Smart hydrogels can improve diabetic wound healing by adapting to wound conditions and providing controlled treatment.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Stress and PCOS together may increase depression and anxiety-like behaviors.

Dermal Wnt/β-catenin signaling is important for the proper size and development of hair follicles.

A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.

Deleting the Hoxc13 gene in frogs shows its crucial role in developing skin structures similar to hair.

The human scalp hair bulb contains different types of melanocytes with varying abilities to produce melanin.

Hidradenitis suppurativa may mainly affect vellus hair follicles, not terminal ones.

The interdigital gland of Vembur sheep shows sex-based differences in size and chemical makeup, possibly affecting communication and disease protection.

Stress hormone corticosterone suppresses hair growth by affecting stem cell activity and Gas6 protein expression.

Restoring EDA and WNT pathways early may help improve skin, hair, and teeth issues in hypohidrotic ectodermal dysplasia.

Hedgehog signaling in skin cells is crucial for hair growth and skin healing, but needs to be balanced to avoid harmful effects like scarring and cancer.

The model better predicts how water-loving and fat-loving substances move through the skin by including tiny pores and hair follicle paths.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

The ovary mainly causes high testosterone in PCO, while the adrenal gland is the main source in IH.

Early neurosteroid changes can alter adult brain function and behavior.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

Neuroactive steroids affect cocaine's rewarding effects through the ς₁ receptor.

Nipple area expansion in mice needs both pregnancy hormones and mechanical strain.