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Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

The ventral matrix is the main source of the nail plate.

The study mapped keratin 15 and 19 genes, aiding future genetic disorder research.

3,4,5-tri-O-caffeoylquinic acid promotes hair growth by activating the β-catenin pathway.

Gene regulatory regions evolve faster than protein coding regions, allowing new gene relationships without changing transcription factors.

The FGF5 gene determines hair length in dogs.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.

Hox genes control hair follicle stem cell regeneration in different body regions.

Potassium channel openers could help treat cardiovascular diseases and asthma but require better targeting to specific tissues for effective use.

A protein called COMP is part of the connective tissue in normal human hair follicles and may be important for hair health.

FLCN helps control iron levels in cells.

Keratin-associated proteins are part of the developing hair fiber cuticle.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Removing HIF-P4H-2 from certain skin cells in mice causes hair loss on the body but not the head.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

OCT is a reliable, noninvasive way to measure hair thickness.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Collagen XVIII is crucial for maintaining tissue structure and function in the brain, kidneys, and hair.

Ptprq has multiple forms that change during inner ear development.

NAC1 controls certain enzymes that reduce root hair growth in Arabidopsis.

The study found nine new hair protein genes in human hair follicles.

Pilomatricomas don't follow the usual hair follicle cell differentiation process.

Collagen XVIII affects wound healing, hair growth, and bone development, with its absence speeding up processes and overexpression causing delays and abnormalities.

Kremen is crucial for proper development and preventing tumors by regulating Wnt signaling.

Ornithine decarboxylase is crucial for human hair growth and the hair cycle.

A specific gene mutation causes a severe skin disorder in a family.