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Mutations in the NR3C1 gene cause a rare condition that affects hormone signaling and can lead to various symptoms, with dexamethasone as a treatment option.

Chronic finasteride use in male rats doesn't strongly cause depression or anxiety due to adaptive stress hormone changes.

Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

Blocking 11β-HSD1 reduces stress hormone damage in hair growth cells.

Cortisone suppresses hair regrowth, especially undercoat hair, while adrenalectomy affects hair growth differently in rabbits and rats.

Patients need better information about the risks of long-term steroid use.

Neonatal allopregnanolone and stress affect behavior differently in adolescence and adulthood.

The COVID-19 pandemic did not significantly change stress levels in preschoolers, but higher-income families' children showed higher stress.

High and low doses of prednisolone helped 62% of children with severe alopecia regrow hair with some weight gain and mild acne as side effects.

Stress increases neurosteroids that help prevent seizures.

Neuroactive steroids affect cocaine's rewarding effects through the ς₁ receptor.

A new gene variant causes glucocorticoid resistance in a mother and son.

Blocking allopregnanolone production in mice makes them more anxious after stress, but this can be reversed with a drug that mimics allopregnanolone.

Steroids may not work well for alopecia areata because of high unoccupied receptors and low thioredoxin levels.

Allopregnanolone is needed for certain brain processing issues caused by D1 dopamine receptor activation.

Large doses of glucocorticoids are not suitable for general use in treating severe alopecia areata due to inconsistent results and risks.

Most adrenal cortex tumors are benign and non-secreting, but proper diagnosis and treatment are important due to the rare possibility of cancer.

Careful patient selection is key for effective pulse corticosteroid treatment in children with alopecia areata.

Women with non-classical congenital adrenal hyperplasia have higher levels of certain steroids, which can be reduced by treatment.

AKR1D1 controls glucocorticoid levels and receptor activity in liver cells.

Targeting 5α-reductase type 1 may help manage Tourette-like symptoms.

Blocking CRF1 receptors improved male hormone levels and reduced testicular tumor size in men with a specific adrenal condition.

Blocking 11β-HSD1 can lessen the harmful effects of glucocorticoids on hair growth cells.

The terrier dog with pituitary-dependent hyperadrenocorticism was successfully treated with mitotane.

Corticosterone makes ethanol more effective at blocking brain processes important for learning and memory.

Ashwagandha may help reduce symptoms of adrenal hyperplasia.

Nonclassic adrenal hyperplasia is a genetic condition that can cause early puberty and fertility problems, treated with specific steroids.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

Viral reactivation is rare at the time of DRESS diagnosis in the U.S.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.