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First Korean case of Cronkhite-Canada syndrome with colon cancer and serrated adenoma.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

lncRNAs significantly influence koi carp skin color.

Proretinal nanoparticles improve skin absorption and reduce irritation of topical retinoids.

KRT14 gene variants cause dermatopathia pigmentosa reticularis, affecting nails, teeth, and hair.

Higher expression of the keratin-associated protein 8.1 gene in Liaoning cashmere goats is linked to finer cashmere fibers.

Patients with a specific genetic variant have more severe alopecia areata and higher recurrence rates.

Different wool coat types in goats are linked to specific gene expressions, which could improve cashmere quality.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Hoxc13 regulates specific hair protein genes on mouse chromosome 16.

Corticosteroids improved symptoms in a man with Cronkhite-Canada Syndrome.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

FLRG and follistatin have different roles in wound healing.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

CDKN2AIP gene is less active in nevus sebaceous, affecting related RNA networks.

NFIC helps rat dental cells grow and turn into bone-like cells.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

circCFAP20DC helps goat ovarian cells grow, aiding follicle development.

CRH can cause hair loss by promoting cell death in hair growth cells.

Lack of Cisd2 disrupts calcium balance in cells, leading to poorly functioning neutrophils.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Mutations in the SRD5A3 gene cause a new type of glycosylation disorder by blocking the production of a molecule necessary for protein glycosylation.

A new sheep gene, KRTAP36-1, may help breed sheep with better wool by reducing prickle factor.

CCCA may be a fibroproliferative disorder, and anti-fibrotic therapies could help.

Nucleic acids trigger chemokine production in skin cells, affecting skin inflammation.

DORN1 receptor affects eATP-induced stomatal changes but not eADP in Arabidopsis thaliana.

Sry may regulate fatty acid metabolism and shows different expression levels in rat tissues.