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Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

African spiny mice can regenerate skin and hair after wounds due to specific tissue mechanics.

Transplanted bone marrow cells actively move, form clusters, and grow after transplantation.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

HoxC genes are crucial for normal hair and nail development.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

Mouse models are essential for studying and improving genetic traits in agriculture.

The C3H/HeJ mouse model is useful for studying and testing treatments for alopecia areata.

A new method can detect mutations in mice by observing changes in hair follicle cells.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

A new mouse model for vitiligo helps study immune responses and potential treatments.

GFP transgenic mice help study cell origins in skin grafts.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Overexpression of COX-2 in mice skin causes abnormal skin and hair development.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

EPI-NCSCs from hair follicles may help treat brain development issues in mice.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.

Two mouse mutants have defective hair cuticle cross-linking.

Deleting the Hoxc13 gene in frogs shows its crucial role in developing skin structures similar to hair.

C57BL/6 mice and SD rats have different sweat gland and hair follicle patterns, useful for skin research.

Injecting certain cells into mice caused hair loss, which was preventable with a specific inhibitor.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Cells from skin and lung can help regenerate hair follicles.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.