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Genetic differences within ethnic groups may affect prostate cancer treatment effectiveness.

A PKP1 gene mutation causes skin fragility and hair loss in Chesapeake Bay retriever puppies.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

Researchers found genes for cysteine-rich proteins that form the protective layer of hair in humans and sheep.

Different gene mutations cause different types of ichthyosis, with some new mutations found.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

High doses of cyproterone acetate are linked to an increased risk of developing meningioma.

Certain gene variations and different levels of BDNF and CRH hormones are linked to vitiligo.

The TIP1 gene is crucial for normal plant cell growth in Arabidopsis.

The peach gene pCTG134 helps control the interaction between auxin and ethylene hormones during fruit ripening.

Some men's prostate tissues have low enzyme levels due to genetic changes, possibly affecting treatment for prostate enlargement.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

A new protein linked to hair strength was identified, aiding in understanding brittle hair conditions.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

The study found two new mutations in a Chinese patient with severe biotinidase deficiency.

The KRTAP7-1 gene is very similar across different cattle and yak breeds and likely plays a role in hair strength and shape.

Mutations in the KRT85 gene cause hair and nail problems.

Gene variant linked to prostate cancer, hormone levels, and hair loss.

Genetic variants in PDE6H and LPAR6 cause vision impairment and hair loss in two Pakistani brothers.

Certain gene variations may protect against skin issues and oxidative stress in women with PCOS.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Potential new drugs for treating PCOS were identified.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

MCHR2 gene duplications may be linked to alopecia areata.

Blocking CXXC5 speeds up diabetic wound healing by improving blood vessel growth and skin repair.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.