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The research found changes in gene expression related to cell death in mouse skin that help understand hair follicle development and skin health.

The SYP123-VAMP727 complex is important for transporting materials that harden the root hair shank in Arabidopsis.

Lower PPARγ levels and specific gene variations are linked to more severe Frontal Fibrosing Alopecia.

RPGRIP1L helps skin cells stick together by blocking PKCβII, which can prevent skin blistering like in pemphigus.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

PDGFC gene may help select goats with desirable curly wool traits.

aPKCλ is crucial for keeping hair follicle stem cells inactive and maintaining normal hair growth.

Certain circular RNAs help cashmere goats grow more hair.

CTCF protein is essential for skin and hair follicle development in mice.

Certain genetic variations in the SHBG gene are linked to an increased or decreased risk of PCOS in Mediterranean women.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

ALRN-6924 can protect hair follicles from chemotherapy damage by temporarily stopping cell division.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Congenital Adrenal Hyperplasia is a genetic disorder with two forms, causing symptoms like early puberty and severe acne, but can be identified through screening and treated with glucocorticoids.

PAK4 is crucial in cancer progression, brain development, and could be a therapeutic target, especially through the PAK4-CREB axis.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

The protein called small nuclear ribonucleoprotein polypeptide E is identified as a cause of a type of hair loss without other symptoms.

Lysine carboxymethyl cysteinate (LCC) protects skin from UVB damage by activating autophagy.

Heart-specific steroid metabolism is crucial in cardiac hypertrophy.

ZmSPL10, ZmSPL14, and ZmSPL26 genes are essential for forming maize structures needed for pollen capture and kernel production.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

The AR gene is linked to male-pattern baldness, TNFSF4 to heart disease, SLC19A3 to BBGD, MCT8 to a syndrome, and segmental duplications to genetic variation.

A specific gene variation increases the risk of high uric acid and cholesterol in young Mexican males.

Certain genes in Iranian sheep are linked to wool production and heat adaptation.

A new therapy for a skin blistering condition has not been developed yet.

Paeoniflorin protects brain cells by involving a specific protein and neurosteroids.