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Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

BST2 is a key marker for hair loss disease alopecia areata.

The B2 genes are crucial for hair growth in rats.

Animal models, especially mice, are essential for advancing hair loss research and treatment.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Diabetes changes keratin in skin and hair, affecting their health.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.

Methamphetamine affects gene expression in rat whisker follicles, with key genes linked to addiction.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Mice with autoimmune hair loss showed signs of heart problems.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

The mouse models are effective for testing new hair loss treatments.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

The BMP2 gene is more active in the early growth phase of Cashmere goat hair and may affect hair regeneration and textile production.

Neuroactive steroids affect cocaine's rewarding effects through the sigma1 receptor.

Pododermatitis in farmed mink is likely caused by an unknown infectious agent with secondary bacterial infection.

A new mouse model helps understand and find treatments for alopecia areata.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Meis2 is essential for whisker development, independent of nerve involvement.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Combining terbinafine and mitotane effectively treated a schnauzer's skin infection and hormone disorder.