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A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Certain genes are linked to the risk of developing Alopecia Areata.

Genetic factors, especially PADI3 gene variants, contribute to CCCA in women of African descent.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

Lack of functional CYLD in mice leads to early aging and cancer.

DAVID syndrome is a condition with immune system and hormone deficiencies, needing early diagnosis to avoid serious complications.

CCCA may involve the PD1/PDL1 pathway and increased caspase 3, leading to permanent hair loss.

A person got uncombable hair syndrome from two copies of chromosome 1 from their mother.

Delta1 is crucial for controlling skin cell growth and preventing tumors in mice.

Genetic variants in specific genes cause central centrifugal cicatricial alopecia.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Vitamin D receptor and hairless protein are essential for hair growth.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

FOXN1 duplication can cause excessive hair growth.

A gene variant increases the risk of a type of hair loss by affecting hair protein production.

Researchers found a genetic region that influences the number of coat layers in dogs.

EBV infection increases a specific keratin variant in carcinoma cells, possibly affecting cell structure and cancer progression.

A genetic marker linked to a type of hair loss was found in most patients studied.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Certain genetic markers, especially the MICA gene, are linked to alopecia areata.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

PTEN was identified as a specific marker for the skin disease cutaneous lupus erythematosus, and it helps increase the expression of harmful type I interferons.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.