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A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

Neutrophils quickly respond to skin injury.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Glycerol may improve wound healing and prevent keloids, a device can measure itch intensity, male pattern baldness is highly heritable, and fumaric acid esters may work for psoriasis by causing cell death in T cells.

Targeting protein S-palmitoylation could lead to new skin disease treatments.

A child with skin and tooth symptoms was found to have a genetic mutation causing cardiocutaneous syndrome, leading to heart problems.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Mutations in the SRD5A3 gene cause a new type of glycosylation disorder by blocking the production of a molecule necessary for protein glycosylation.

A patient with a new PLEC gene mutation showed symptoms of both muscular dystrophy and myasthenia gravis, which improved with steroid treatment.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Local inflammation worsens autoimmune skin conditions by increasing antibody buildup.

NG2 is crucial for normal skin and hair development in mice.

A rare genetic variant linked to skin cysts was found in blood DNA, suggesting its role in cyst formation.

The study created hair-bearing skin models that lack a key protein for skin layer attachment, limiting their use for certain skin disease research.

The hydrogel patch helps heal diabetic wounds by releasing a healing agent in response to harmful molecules and improving skin regeneration.

KLHL24-mutant stem cells help understand skin and heart disease.

Corneal health relies on cell migration and cooperation with the lacrimal gland, not Bmi1+ cells, and Eda gene mutations can cause dry eye disease.

A boy with Sjogren-Larsson syndrome has skin and muscle symptoms due to a specific enzyme deficiency.

Loss of OGG1 increases skin inflammation and auto-antibodies in lupus.

New genes linked to ichthyosis were found, but there's still no cure; treatment options are improving.

The absence of functional sebaceous glands causes hair follicle destruction and scarring alopecia.

Deep dermal tissue dislocation injury in pigs leads to thicker fibrotic tissue and increased type III collagen, affecting skin repair.

The PER3 rs772027021 SNP may cause mild skin pigmentation changes in a new subtype of dyschromatosis universalis hereditaria.

Human dermal papilla cell vesicles can reduce skin fibrosis in mice.

A rare gene mutation causes skin fragility and itching without affecting hair or nails.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

Meis2 is essential for touch sensation and nerve function in mice.

MCP@G improves diabetic wound healing by reducing stress and promoting tissue repair.