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A special hydrogel helps stem cells heal wounds better by boosting growth factors.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

A peptide from hair follicle stem cells promotes hair growth by activating specific skin cells.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Epidermal β-catenin activation changes the dermis by signaling different fibroblast types.

Epiprofin helps cells grow in developing teeth, hair, and limbs.

Using dermal papillae cells and keratinocytes in skin substitutes speeds up healing and helps form hair follicles and glands.

Apoptosis is essential for human skin development and forming a functional epidermis.

The study concludes that certain domains in Clostridium histolyticum enzymes are structurally unique, bind calcium to become more stable, and play distinct roles in breaking down collagen, with potential applications in medicine and drug delivery.

The research maps the complex development of early mouse skin, identifying diverse cell types and their roles in forming skin layers and structures.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

PDGF signaling may play a role in hair growth cycle regulation.

BMP signaling is essential for the development of touch domes.

uPA helps hair follicle cells grow.

HSP90 and lamin A/C are crucial for hair growth and could be targets for treating hair loss.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

Reducing PIEZO1 speeds up wound healing.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

iPSC-derived stem cells on a special membrane can help repair full-thickness skin defects.

Horse skin stem cells combined with platelet-rich plasma improve skin healing.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

Mutations in the DSG4 gene can cause a rare hair disorder similar to monilethrix.

CD133+ cells are crucial for hair growth.

Fibroblast state switching is crucial for skin healing and development.

Cathepsin E is crucial for normal skin cell differentiation and development.

DPP4 is important for scarring and skin regeneration, and managing its activity could improve skin healing treatments.

Keratins help protect cells, aid in cancer diagnosis, and influence cancer behavior and treatment.