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Mutations in the SRD5A3 gene cause a new type of glycosylation disorder by blocking the production of a molecule necessary for protein glycosylation.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Dexamethasone reduces inflammation and delays skin cell changes caused by TPA.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

Multiple mouse desmoglein 1 isoforms have distinct roles in skin and hair development.

Sesn2 protects inner ear hair cells from damage by regulating certain cell survival pathways.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

Secukinumab reduces immune activity in hidradenitis suppurativa skin.

Kdm6b is crucial for skin cell differentiation.

Finasteride irreversibly affects human steroid 5α-reductase 2, providing insight into its catalytic mechanism and disease-related mutations.

Human and mouse TGase3 enzymes are similar but differ near the activation site, crucial for their function in skin and hair development.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Anti-Ku-positive patients often have muscle weakness and autophagy plays a key role in their condition.

Peptide drugs now target hard-to-reach proteins more effectively and specifically.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

A new method helps find proteins in hair to identify fetal growth issues.

The BUZZ gene is important for root hair growth and overall root structure in the plant Brachypodium distachyon.

Upadacitinib may effectively treat twenty-nail dystrophy without causing pain.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Blocking 11β-HSD1 can lessen the harmful effects of glucocorticoids on hair growth cells.

New lipid/fiber microplexes improve mRNA therapy for degenerative diseases by enhancing cell function and treatment effectiveness.

Lysosomal proteases and cytoplasmic enzymes help hair follicle cells develop and differentiate.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

Desmosomes are crucial for skin and heart development, and JNK may help regulate them.

Overexpressing thrombospondin-1 in mice skin prevents UVB-induced skin damage.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

A mutation in the human hairless gene causes alopecia universalis.