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Hair and nail cells share similar proteins, indicating a common differentiation pathway.

Epithelial cells from young rat hair follicles grow and form aggregates in culture, but don't produce hair keratin proteins.

Type II keratin genes are crucial for hair follicle differentiation and have a conserved structure and expression pattern.

Rac1 is essential for proper hair structure and color.

Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.

Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

Hair differentiation starts earlier than thought, involving multiple type-II keratins.

hHbl gene is active in hair shaft cells and some pilomatricomas.

S100A3 protein is crucial for hair shaft formation in mice.

Mouse high-glycine/tyrosine proteins have distinct patterns in hair follicles, peaking at specific hair cycle days.

Keratin gene regulation is similar across mammals, affecting hair follicle differentiation.

A unique hair tumor with a rippled pattern was identified, showing incomplete differentiation and unusual cell arrangements.

NuMA-microtubule interactions are vital for proper skin structure formation and function.

Pilomatricomas don't follow the usual hair follicle cell differentiation process.

Hair proteins change location and structure as hair cells mature.

Pili annulati is caused by a protein metabolism disorder affecting hair structure.

CIP/KIP proteins help stop cell division and support hair growth.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Hair follicle stem cells are similar to mesenchymal stem cells and can become neural-like cells under certain conditions.

Hair follicle stem cells are similar to bone marrow stem cells but are better for fat cell research.

The research helps understand how stem cells turn into hair follicle cells.

Activin A and follistatin control when hair cells develop in mouse ears.

Activin A and follistatin control when ear hair cells form in mice.

Activin A promotes ear hair cell development, while follistatin delays it.

Nestin-expressing hair follicle cells may be useful for nerve repair and regeneration.

Cathepsin L is essential for normal hair growth and development.

ES cells can be turned into hair follicle cells in a lab setting.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Activating β-catenin in certain skin cells speeds up hair growth in mice.