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Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Blocking a specific enzyme might help treat obesity and diabetes, but more research is needed to ensure it's safe.

Iron chelators can effectively reduce hair loss and improve hair growth in autoimmune alopecia.

The human type II 5α-reductase gene has a specific structure important for understanding certain medical conditions.

PCOS patients have higher LDH and lower cortisol, dopamine, zinc, and vitamin D3, which may contribute to their symptoms and obesity.

Hirsute women have lower type 2 17β-HSD enzyme levels, which improve with treatment.

4-(4-Phenoxybenzoyl)benzoic acid derivatives can both increase and decrease certain types of reactive oxygen species, and may be relevant to hair loss.

The enzyme system in hair follicles is similar to the liver's and is inhibited by certain substances.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The research gives new understanding on how human steroid 5α-reductases work and how drugs like finasteride inhibit them, which could help in creating new drugs.

Finasteride safely treats enlarged prostate and male-pattern baldness.

Selenium in proteins helps prevent over-oxidation and supports chemical reversibility.

OR2AT4 helps reduce aging and cell damage in human skin cells.

The human type II 5α-reductase gene, linked to certain male health conditions, has a specific structure and low similarity to other related genes.

Tetrathiomolybdate reduces hair growth marker in skin cells by boosting harmful oxygen molecules, but effects can be reversed.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

DHEA boosts bone cell growth and differentiation in elderly stem cells.

Increased HSD11B1 enzyme expression is linked to higher body fat and insulin resistance.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Minoxidil reacts to nitrosation 7 times more than phenol, mainly due to its -NH₂ groups, leading to the creation of N-nitrosominoxidil.

Superoxide dismutase (SOD) can prevent hair graying in mice.

Deuterium oxide extends the hair cycle duration in mice without changing hair structure.

Finasteride blocks deoxycorticosterone's anticonvulsant effects in infant rats, but indomethacin doesn't.

The activity of a specific rat enzyme in the prostate and epididymis is highly dependent on the acidity level.

Liposomes and iontophoresis effectively deliver protective enzymes into the skin against UV damage.

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

A new mutation in the HJV gene was found in a young woman with juvenile hemochromatosis, causing unusual symptoms like secondary hypothyroidism.

The structure of SRD5A reveals how it reduces steroids, aiding drug design for related health conditions.

Dihydrotestosterone (DHT) is crucial for conditions like male-pattern baldness and acne, and measuring a byproduct, androstanediol glucuronide, is a better way to assess DHT's effects than DHT blood levels.

D'orenone stops root hair growth by disrupting auxin transport, but adding auxin can reverse this.