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Finasteride and dutasteride's effects are mainly due to target binding saturation and slow enzyme turnover.

Diphencyprone initially increases mouse hair growth, then slows it, possibly due to changes in specific protein levels.

Mutations in certain receptors can cause diseases and offer new treatment options.

Prostate cells have proteins that bind to specific hormones, which can increase protein production when activated by these hormones.

HDAC inhibitors help brain cells grow and improve brain function.

Changing neuroactive steroid levels early in life can affect how adult rats respond to alcohol's stimulating effects.

Finasteride changes androgen receptor location in rat epididymis without altering tissue structure.

Dihydrotestosterone increases the activity of an enzyme in pubic skin cells that converts testosterone to dihydrotestosterone.

PDGF signaling may play a role in hair growth cycle regulation.

Certain enzymes react strongly with some hormones in rat skin during hair growth, mainly in sebaceous glands and hair sheaths.

New steroids were effective in blocking male hormone receptors in hamster prostates.

Some cannabinoid derivatives may be more effective than current drugs at targeting proteins relevant to prostate cancer treatment.

Muscarinic acetylcholine receptors in skin cells help regulate and promote hair growth.

The document confirms the structures of major metabolites of the CRTh2 antagonist Setipiprant and identifies minor metabolites.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Finasteride's effect on hair loss and prostate enlargement depends on its binding to an enzyme, with maximum impact at 0.2 mg dose.

Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

Higher SRD5A2 expression predicts better response to finasteride in treating urinary symptoms.

Dexamethasone may influence hair growth by altering estrogen receptor activity in hair cells.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Certain progesterone derivatives can inhibit enzymes and reduce androgenic activity, potentially affecting prostate growth.

Reducing 5α-reductase activity helps endometrial cells differentiate, aiding pregnancy.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Some prostate cancers have gene changes that may affect treatment with certain drugs.

KCNQ potassium channels help control the sensitivity of touch receptors in the skin.

Early androgen exposure affects hormone receptor expression in adult female rats' brains, but not in males.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.