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Accurate diagnosis of cicatricial alopecias requires thorough scalp examination and multiple biopsy techniques.

Gene differences affect finasteride side effects in men with hair loss.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

The gene therapy showed significant wound healing and was safe for treating severe RDEB.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

UC-MSC-Es help human hair cells grow.

The ZIP13 variant is linked to abnormal hair quality.

ULBP3 could be a marker for diagnosing alopecia areata incognita and may be linked to its cause and development.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Escin may help treat hair loss by boosting a specific cell growth pathway.

BMPR1a-ECD reduces wrinkles much more effectively than retinoic acid.

The study concludes that mutations in the AEBP1 gene can cause a form of Ehlers-Danlos syndrome and should be considered in diagnosis.

The ABI1 gene contributes to prostate cancer progression and treatment resistance.

Plakophilin 1 helps control skin cell immune responses to prevent excessive inflammation.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

Keratin is crucial for skin barrier formation and affects mitochondrial function.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Vitamin D can reduce skin inflammation caused by UV rays by enhancing cell "fitness" and skin repair.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

The enzyme PA-PLA1α is important for proper hair follicle development.

PADI4 enzyme slows down cell growth in developing hair follicles.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.