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People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

DHT deficiency may disrupt rat epididymis function by affecting estrogen receptors.

The vitamin D receptor can work without its usual activating molecule.

A mutation in the EDNRA gene causes Oro-Oto-Cardiac syndrome, affecting face and heart development.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

A PKP1 gene mutation causes skin fragility and hair loss in Chesapeake Bay retriever puppies.

TRIV-509 quickly improves skin barrier and cell health in atopic dermatitis.

KID syndrome should be reclassified as an ectodermal dysplasia.

EVAL membranes help create cell structures that can regrow hair follicles.

LPA3 signaling in the uterus is crucial for placental formation and fetal development.

Delta1 is crucial for controlling skin cell growth and preventing tumors in mice.

Scalp hair follicle cells help protect and heal skin in certain skin conditions.

A new EDA gene mutation was found in a Chinese family with a specific skin disorder.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

A chromosomal change may cause ectodermal dysplasia and developmental issues in a child.

sPLA2-IIE is crucial for normal hair follicle structure and skin health.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

New LPA receptors (LPA4, LPA5, LPA6) have diverse roles in the body.

Retinoic acid can change skin development, like turning scales into feathers or forming glands.

cDermo-1 causes dense skin, feathers, and scales in chickens.

Vitamin D receptor helps control skin cell growth and hair development.

Human hair follicles contain a complex network of prostanoid receptors that may influence hair growth.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

EPDS and MS might share an immune-related cause.

BMP-2 activates the Dlx3 gene in mouse skin cells, important for hair and skin development.

TEDAR is crucial for skin cell differentiation and barrier formation.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Vitamin D receptor is essential for proper skin healing after injury.