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A patient with a PLEC mutation has epidermolysis bullosa, muscular dystrophy, and myasthenia gravis, which improved with steroid treatment.

Blocking a protein called prostaglandin D2 might help treat hair loss.

DEC cells show promise as a safe and effective treatment for Duchenne muscular dystrophy.

Higher levels of the DP2 receptor may lead to hair loss.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

The role of certain genes in skin cell development may be linked to psoriasis, but the connection is unclear.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Vitamin D receptor is essential for healthy bones and skin.

LPA3 signaling in the uterus is crucial for placental formation and fetal development.

Vitamin D receptor is crucial for skin wound healing.

Phospholipids help plant proteins move by regulating receptor interactions.

Dermal EZH2 controls skin cell development and hair growth in mice.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

Epigenetic mechanisms control skin development by regulating gene expression.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

Mutations in specific genes cause different types of ectodermal dysplasias.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Delta opioid receptors help regulate touch sensation by reducing neurotransmitter release in the spinal cord.

Carriers of a specific gene mutation have subtle skin changes without visible symptoms.

Vitamin D receptor helps control hair growth genes in skin cells.

TCDD reduces EGF receptor activity and causes various developmental changes in animals.

Desmosomal adhesion is essential for healthy skin structure and function.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

DHT deficiency may disrupt rat epididymis function by affecting estrogen receptors.

The vitamin D receptor can work without its usual activating molecule.

A mutation in the EDNRA gene causes Oro-Oto-Cardiac syndrome, affecting face and heart development.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.