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Certain changes to the B-ring of androgen receptor ligands can increase their effectiveness for potential treatments of muscle and bone conditions.

RXR agonists may promote hair growth in humans.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

PDRN from trout sperm helps skin and hair regeneration but is costly and complex to produce.

Low ERCC3 gene activity is linked to non-pigmented hair growth.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

The mineralocorticoid receptor temporarily affects mouse skin development, but the glucocorticoid receptor has a more lasting impact.

Edar signaling is crucial for controlling hair growth and regression.

A vitamin D receptor mutation causes rickets and affects immune responses.

GFRα2 is essential for controlling neuron size but not for target innervation in certain sensory neurons.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

New probes were created to effectively measure specific enzymes involved in fat metabolism, which could help develop new drugs.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

OsUEV1B protein is essential for controlling phosphate levels in rice.

The activity of a specific rat enzyme in the prostate and epididymis is highly dependent on the acidity level.

Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

Three specific genetic variants cause severe skin issues in children with EBS, highlighting the need for early genetic screening.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

RNase L hinders hair follicle regeneration by altering immune signals.

The new model helps understand and develop treatments for genetic skin disorders like AEC.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

ceRNA networks offer potential treatments for skin aging and wound healing.