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NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Mutations in the p63 gene affect skin adhesion, barrier integrity, and hair growth.

Mice with human skin protein K8 had more skin problems and cancer.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

Loss of TET2 increases the risk of skin and oral cancer.

Erlotinib can cause acne-like skin issues, needing early treatment and possible dosage adjustments.

New mutations in KRT83 and KRT86 are linked to the hair disorder monilethrix.

Overexpressing PKCepsilon in mice reduces papillomas but increases carcinomas.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Keratinocyte growth factor significantly alters skin and tissue development.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

A new mutation in the KRT86 gene causes monilethrix in a Han family.

Epidermal Growth Factor helps hair follicle cells grow and move by activating a specific cell signaling pathway.

A new genetic region, 17q21.31, is linked to higher ovarian cancer risk.

cgVEGF164 boosts hair follicle growth in mice.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

HPV8 causes skin cancer by expanding specific skin stem cells.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

Dabrafenib can cause skin growths and sometimes low-grade skin cancer.

High amphiregulin in the skin is a bad sign for acute graft-versus-host disease.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

The most common sign of aging at the gene level is more Ectodysplasin A2 Receptor (EDA2R) being made.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

ERK activation spreads between cells, influencing cell division and wound healing.

A man's bald spot grew hair after starting cancer treatment with gefitinib.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

ROR-alpha may increase the growth of certain breast cancer cells by boosting aromatase, which could affect breast cancer prognosis.