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A specific genetic change is linked to mental disorders, intellectual disability, and possibly autoimmune disease in a family.

A new mouse mutation causes skin and hair defects due to a gene change.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Blocking autophagy increases survival of inner ear hair cells exposed to gentamicin.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

ERULUS is crucial for root hair growth by controlling calcium levels.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

Ectodysplasin-A1 is crucial for developing hair, teeth, and glands.

mIGF-1 in skin cells speeds up wound healing and hair growth in mice without harmful effects.

Some leukemia treatments can cause skin reactions similar to keratosis pilaris.

Keeping human skin stem cells is easier with low temperatures and mTOR inhibition.

Mouse hair keratins mHa1 and mHb4 can't form a strong network on their own in cells.

The hamster polyomavirus middle T antigen is linked to tumors in hamsters and associates with a specific tyrosine kinase.

A gene called Taqpep affects cat coat patterns like stripes and blotches.

Edar and Eda proteins are crucial for proper tooth development.

A rare skin condition with cysts was found on a 5-year-old boy's scalp.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

A new mutation in the KRT86 gene causes monilethrix in a Han family.

Monilethrix is a genetic hair disorder affecting hair shape, seen in two brothers.

p63 controls Satb1 to help skin develop properly.

Personalized treatment based on detailed tumor analysis successfully managed and reduced the patient's aggressive hair follicle cancer.

A new therapy for a skin blistering condition has not been developed yet.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.