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Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

Some anticonvulsant drugs can cause skin reactions, ranging from mild to severe, and managing these reactions is important for patient care.

The dietary supplement significantly improved skin, nails, and hair in older adults.

Dermal papilla cells help wounds heal better and can potentially grow new hair.

Cornification evolved from keratinization in vertebrates, with differences between mammals and sauropsids.

TCF/Lef1 activity is essential for proper skin cell development and renewal.

CRISPR-based tools improve understanding and treatment of skin development and conditions.

EGF–FGF2 helps mouse stem cells grow and become more like nerve cells.

Sebaceous glands can form new hair follicles when activated, but hair follicle bulges cannot.

Mimicking fetal wound environments may enable scarless healing in adults.

Necl2 affects skin cell behavior and slows wound healing.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Stem cells from elderly skin can become neurons, offering potential for brain therapy.

Autophagy is essential for proper skin cell development and function.

Reprogramming adult fibroblasts may enable scar-free healing.

Vitamin A helps skin stem cells decide their function, aiding in hair growth and wound repair.

Vitamin D receptor is crucial for skin wound healing.

Epidermal stem cells show promise for skin repair and regeneration.

Sox2-positive cells determine specific hair follicle types in mammals.

Hair growth can be stimulated by combining certain skin cells, which can rejuvenate old cells and cause them to specialize in hair follicle creation.

Reducing Flightless I protein improves wound healing by activating skin stem cells.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Specific cross-linkages help make hair proteins stable and strong.

The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

Epidermal stem cells show promise for treating orthopedic injuries and diseases.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Hair follicle stem cells can form both hair follicles and skin.