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Some drugs, especially biologics, can cause skin reactions that look like other skin diseases, and stopping the drug usually helps clear up these reactions.

Piperonylic acid speeds up wound healing by reducing inflammation and boosting collagen.

Apoptosis is essential for human skin development and forming a functional epidermis.

The skin is the largest organ, protecting the body, regulating temperature, and producing hormones.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

The skin's layers protect, sense, and regulate the body's internal balance, but can be prone to cancer.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Merkel cells may have roles in sensing magnetic fields, creating fingerprints, Reiki energy healing, passing on environmental information to offspring, and influencing hair shape.

Chitosan nanofiber scaffolds improve skin healing and are promising for wound treatment.

Growth factors are crucial for hair development and could help treat hair diseases.

Deleting MED1 in skin cells causes hair loss and skin changes.

Melatonin stimulates the skin components of ram's scrotum during their non-breeding season.

Porphyra-334 may help reduce wrinkles and promote hair growth.

Touch dome keratinocytes in adult skin have traits of different skin cell types.

17β-estradiol may help hair growth by increasing cannabinoid receptor type 1.

Removing Lrig1-positive stem cells in mice causes temporary loss of sebaceous glands.

Electrical epilation damages hair follicles and surrounding skin, likely preventing hair regrowth.

Monotreme hair structure and protein distribution are similar to other mammals, but their inner root sheath cornifies differently, suggesting a unique evolution from reptile skin.

Norfolk Terriers have a genetic skin defect causing scaling and blisters due to a keratin issue.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Skin has specialized touch receptors that can tell different sensations apart.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

Scientists created a long-lasting stem cell line from human hair that can turn into different skin and hair cell types.

YAP and TAZ proteins control skin cell growth and repair.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

SCD-153 shows promise as an effective topical treatment for alopecia areata.