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Corneal cells can transform into hair-producing skin cells when exposed to certain signals.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Blocking certain proteins can reduce skin inflammation caused by cancer treatment.

K16 can partially replace K14 but causes hair loss and skin issues.

High proliferation and cell delamination drive early skin development, while later stages may not rely on cell division orientation.

MCSP may help identify and regulate skin stem cells, affecting hair growth and regeneration.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Transamidases are present in the epidermis but their exact role is unclear.

Mice with human skin protein K8 had more skin problems and cancer.

Overexpressing the MSX-2 gene in mice causes skin and hair growth issues.

Cholesteatoma shows abnormal and increased EGF receptor expression, indicating its rapid growth.

Trichohyalin is also found in the outer layers of normal human skin.

Progerin affects cell shape but not hair or skin in mice.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Increasing calcium sensing receptor speeds up skin and hair development in mice.

Stem cells in the skin help with healing and can also lead to cancer.

Epidermal Growth Factor helps hair follicle cells grow and move by activating a specific cell signaling pathway.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

PAD type III enzyme is specific to rat skin and hair follicles.

A form of vitamin E promotes hair growth by activating a specific skin pathway.

Porcine skin cells are useful for studying wound healing and cancer.

Atoh1 expression can create new Merkel cells in the skin.

Deleting MED1 in skin cells causes hair loss and skin changes.

The gelatin/β-TCP scaffold with nanoparticles improves wound healing and skin regeneration.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

Ephrins slow down skin and hair follicle cell growth.

Neuregulin3 affects cell development in the skin and mammary glands.