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TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Vitamin A treatment reduced abnormal cell growth and improved skin conditions in rats with tumors.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

Ginsenoside Rg3 may help hair growth by increasing a growth-related protein in hair cells.

Blocking EGFR in mice causes hair loss and skin changes.

A specific DNA region controls skin cell gene expression by working with certain proteins.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Sex steroids influence skin appendage function and neoplasm development, with changes in AR and ERalpha in certain gland neoplasms.

Eyelid cells share signaling components but differ in pathway activity.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Endoglin is important for proper hair growth cycles and stem cell activation in hair follicles.

Beta-catenin is crucial for skin cell growth, development, and cancer formation.

The gene therapy showed significant wound healing and was safe for treating severe RDEB.

The drug showed promise in treating renal cell carcinoma with manageable side effects.

Erk signaling helps zebrafish fins regrow to the right size by using memory of the original size.

Enobosarm may effectively treat androgen receptor-positive breast cancer with fewer side effects.

Erdr1 could be a new marker for diagnosing hair loss.

Cannabinoid receptor-1 signaling is essential for the survival and growth of human hair follicle stem cells.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Edar signaling is crucial for proper hair follicle development and function.

A mutation in the EDNRA gene causes Oro-Oto-Cardiac syndrome, affecting face and heart development.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

Removing EGFR in skin causes inflammation and abnormal hair growth.