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Cannabinoid receptor-1 signaling is essential for the survival and growth of human hair follicle stem cells.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

Increased Stat3 activity reduces hair follicle stem cells and boosts other stem/progenitor cells.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

The E2 protein affects gene activity in hair follicles of mice.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

Hedgehog signaling is crucial for hair development, cadherins affect cell adhesion, neutrophils play a role in skin lesions, and BP230 autoantibodies impact skin stability.

The extracellular matrix affects where tumors can start in the body.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

Eyelid cells share signaling components but differ in pathway activity.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

Increasing EGR1 levels makes hair root cells grow faster.

Notch signalling helps skin cells differentiate and prevents tumors.

Fibroblasts from healthy donors can prevent changes seen in recessive epidermolysis bullosa simplex.

Bcl-2 helps hair regeneration but can also increase cancer risk.

Increasing calcium sensing receptor speeds up skin and hair development in mice.

ERULUS controls root hair growth by regulating cell wall composition and pectin activity.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Blocking certain proteins in mouse skin can reduce and shrink skin tumors.

CD34 is crucial for skin tumor development in mice.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

Type I keratin genes are closely linked to the rex locus on mouse chromosome 11, affecting hair development.

The synthetic retinoid EC23 thickens skin and promotes hair growth more effectively and with a lower dose than natural retinoids.