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The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

The three estrogen receptor genes are highly expressed in zebrafish neuromasts during development.

Dermal EZH2 controls skin cell development and hair growth in mice.

Lack of KSR1 stops certain skin tumors in mice.

ILC1-like cells can cause alopecia areata by themselves.

Dermal EZH2 controls skin cell growth and differentiation in mice.

New cancer treatments show promise in reducing tumor growth and improving skin regeneration in mice.

BerEP4 and CD34 staining can help tell apart tricholemmoma from basal cell carcinoma.

RXRα is crucial for hair growth and skin cell function.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

RXR and RAR proteins in skin may help with cell growth, hair growth, and gland function.

Activating ER-β, not ER-α, improves skin cell growth and wound healing.

EGFR helps control how hair grows and forms without needing p53 protein.

The FRZB gene slows hair growth in rabbits.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

Brg1 is crucial for keeping hair follicle stem cells and repairing skin, working with the Sonic Hedgehog pathway to promote hair growth.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

BMP receptor IA is essential for proper hair cell differentiation in mice.

c-Kit is important for heart regeneration and cancer development.

Changing YBX1 protein activity affects skin stem cell function and aging.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

CRABP2 helps increase the growth of cells important for hair growth by activating a specific growth pathway.

XEDAR triggers a specific signaling pathway in cells.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

EGFR helps hair follicles transition properly by controlling Stathmin levels.