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Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Targeting JAK-STAT1 can reduce inflammation and promote hair growth in conditions linked to EGFR deficiency.

Testosterone increases cell growth and reduces IL-6 in gum cells.

BTNL2 helps protect hair follicles from immune attacks.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Growth factors can either promote or inhibit hair follicle cell growth and collagen breakdown.

EGFR inhibitors can cause yellowish skin eruptions.

EGFR is essential for organized skin nerve growth and branching.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

Activating the GDNF-GFRα1-RET signaling pathway could potentially promote skin and limb regeneration in humans and could be used to treat hair loss and promote wound healing.

EGF, IGF-I, and IGF-IR genes are crucial for mink skin and hair growth.

The assay quickly identifies substances that increase or decrease blood vessel growth.

15d‐PGJ2 cream protects against UVB-induced skin damage in mice.

CD4+ skin cells may be precursors to basal cell carcinoma.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

Epidermal growth factor helps hair regrow after chemotherapy.

Hair follicle cells produce VEGF, which can promote blood vessel cell growth and movement.

S100A6 is important for cell functions and can help diagnose and treat diseases.

Laminin-511 is essential for proper melanocyte movement and development in mice.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

FGFs-4, -8, and -9 have overlapping roles and are repeatedly used in tooth development.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

Mouse skin fibroblasts vary in function and adaptability based on their environment.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

Recurrent NICF is a rare skin condition with unclear causes, involving follicle inflammation and crystal deposits.

Aryl hydrocarbon receptor overexpression found in miniaturized hair follicles in female hair loss.

FERONIA regulates plant growth, pollen interactions, and sugar signaling.

CD44 variant changes start alopecia areata, but don't maintain it.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Collagen XVII and CD151 affect cell movement, with CD151 inhibiting migration when bound to integrins.