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LGR5 is a common marker of hair follicle stem cells in different animals and is important for hair growth and regeneration.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

EGF and FGF signaling stops hair follicle development in mice.

Fatty acid transport protein 4 is essential for skin and hair development.

IGFBP5 may be a potential target for Parkinson's treatment by reducing neuron death.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Mouse models help study genetic skin diseases.

PIKFyve is essential for normal platelet function and its deficiency causes organ issues and macrophage infiltration.

The study created a mouse model that survives longer and shows fewer symptoms of pemphigus vulgaris.

C57BL/6 mice and SD rats have different sweat gland and hair follicle patterns, useful for skin research.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

The mutation helps mice handle heat better without affecting hair growth.

The G(S) alpha subunit gene may help start hair follicle growth in newborn mice.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

Reduced EGFR signaling delays hair cycle and reduces fat growth, but hair development remains normal.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.