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The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

Weak cell junctions disrupt hair follicle stem cell rest.

Nonpalmoplantar skin cells can be made to express keratin 9 by interacting with palmoplantar fibroblasts.

Nexin 1 may help control hair growth.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Mutations in specific llama genes may affect fiber quality for textiles.

Nephronectin helps attach muscle cells to hair follicles.

JAM-A modification speeds up skin wound healing by boosting fibroblast growth.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

Collagen XVII is crucial for skin cell growth and nail health.

KRT72 gene helps form hair.

Scalp hair follicle cells help protect and heal skin in certain skin conditions.

Early and accurate diagnosis is crucial for managing rare genetic disorders like this localized variant of junctional epidermolysis bullosa.

Understanding the nanoscale structure of skin fibrosis can improve knowledge of wound healing and tissue regeneration.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

ALKBH5 helps wounds heal faster by stabilizing PELI2 mRNA.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

bFGF delays hair growth in mice.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

Human growth factor 20 can boost mouse whisker growth.

Fetal skin biopsy can help diagnose protein-related disorders before birth.

Focal palmoplantar callosities may help diagnose non-Herlitz junctional epidermolysis bullosa.

Mouse skin fibroblasts vary in function and adaptability based on their environment.

Three genes, BMP4, POSTN, and WNT5A, may help treat keloids.

Researchers found genes for cysteine-rich proteins that form the protective layer of hair in humans and sheep.

A genetic marker linked to a type of hair loss was found in most patients studied.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.