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FERONIA and LRX proteins help control cell growth in plants by regulating vacuole expansion.

Researchers found a new mutation in the FERMT1 gene in a Spanish family with Kindler syndrome.

Folate receptor β helps suppress the immune system in macrophages and affects cancer growth and hair health.

Disrupting the FGF5 gene in rabbits leads to longer hair by extending the hair growth phase.

Frontal Fibrosing Alopecia involves disrupted cholesterol pathways, fibrosis, and increased mast cells.

A gene variation is linked to hair thickness in Asians.

The flaky skin mouse mutation is a natural model for studying human psoriasis.

Zfp157 is active in many mouse tissues during development and in specific adult cells.

Fgfr2b helps maintain healthy skin and prevent cancer.

Daughters with affected mothers may develop frontal fibrosing alopecia early.

Frontal fibrosing alopecia is linked to four genetic areas, especially the HLA-B*07:02 allele.

Targeting the TNFRSF1B gene may help treat hair loss.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.

Researchers identified a new variant of the FGF5 gene in sheep that affects hair length.

A protein called FERONIA helps control root hair growth in response to cold and low nitrogen by activating nutrient-sensing pathways in a plant called Arabidopsis.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

PRSS35 enzyme may help start skin tumors and could be a target for cancer treatment.

FGF5 spliceosomes inhibit rabbit hair growth by affecting gene expression.

DNAJB9 cfRNA could help diagnose and treat female hair loss.

SFRP2 and PTGDS may be key factors in female hair loss.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Oral contraceptive use may increase the risk of frontal fibrosing alopecia in women with a specific CYP1B1 gene variant.

A 46-year-old man was diagnosed with frontal fibrosing alopecia, a condition usually seen in postmenopausal women.

FFA's causes may include environmental triggers and genetic factors.

Certain genes controlled by OVOL1 are crucial for creating new hair follicles.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.