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Pregnancy can change skin disease severity, with some conditions improving and others worsening, and treatment should balance benefits and fetal safety.

Pregnancy can cause specific skin conditions that need correct diagnosis and treatment to protect both mother and baby.

Pregnancy can cause various skin changes, including pigmentation, acne, and stretch marks, and may affect preexisting skin conditions.

The skin condition PUPP usually gets better on its own after giving birth and is unlikely to happen again in future pregnancies.

Pregnancy can cause skin changes and may affect pre-existing skin conditions, with some treatments not safe for use during pregnancy.

Oral contraceptives can affect skin and hair, improve acne, but may cause pigmentation, dermatitis, and other conditions.

Herpes gestationis is linked to certain antigens, atopic eczema affects T cell populations and may be eased by breastfeeding, higher doses of anti-androgen treatment can improve androgenic alopecia, topical minoxidil increases hair thickness, long-term methotrexate therapy can cause liver fibrosis in psoriasis patients, and Lichen Sclerosus et Atrophicus patients aren't at higher risk for autoimmune disorders.

PCOS increases risks of gestational diabetes, hypertension, and caesarian deliveries in pregnancy.

Fetal sheep develop skin nerve pathways and responses to touch and heat between 75 and 134 days of gestation.

Being overweight or obese before pregnancy increases the risk of having a C-section and a larger-than-normal baby in Chile's public health system.

Resveratrol during pregnancy didn't harm male rat development.

Understanding fetal skin development helps diagnose congenital skin diseases.

Overweight or obese women before pregnancy have lower iron levels in their serum and hair during early pregnancy.

Melanocyte precursors in human fetal skin follow a specific migration pattern and some remain in the skin's deeper layers.

Fetal hair follicles have melanocytes with melanosomes at different stages, which are broken down into pigment particles in keratinocytes.

Red Sokoto goat fetuses develop firmer, pigmented skin with hair follicles as they grow.

First-time mothers had higher stress hormone levels in late pregnancy than mothers who had given birth before.

Canine fetal hair follicle stem cells show pluripotency, with higher S100 protein expression at 40 days.

Free fatty acids may increase androgen production, potentially contributing to polycystic ovary syndrome.

Carbohydrates are crucial for skin development and may help understand skin conditions.

Hydrogen peroxide can cause scars by changing healing processes and increasing certain protein levels.

A baby girl's hair turned straight on one side due to a neck tumor and surgery, but it returned to curly as she recovered.

Gene mutations can cause problems in male genital development.

Fibromodulin might help reduce scarring if increased in adult wounds like in fetal skin that heals without scars.

Exposure to finasteride in the womb caused lasting reproductive issues in male rats.

Stem cells in developing hair follicles move to specific areas as they mature.

Finasteride, when given in high oral doses to pregnant monkeys, caused genital abnormalities in male fetuses, but not in female fetuses or those exposed to intravenous finasteride.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

5alpha-reductase 2 is crucial for proper male brain development and sexual differentiation.

Keratin 2e shows a unique pattern in developing fetal skin, different from other keratins.