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Certain defective glucocorticoid receptor mutants move faster inside cell nuclei and work less effectively.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Alkaline Ceramidase 1 prevents early hair loss in mice by keeping hair follicle stem cells balanced.

The STRIPAK complex is crucial for skin cell organization and creating a functional skin barrier.

ASLNC168501 may help treat hair loss by boosting hair follicle stem cell activity.

Melanocyte stem cells can become melanoma, resembling human melanoma.

Mouse models help target specific genes in lymphatic cells for research.

Four transcription factors can convert mouse cells into hair cell-like cells, aiding hearing loss research and treatment.

Meis1 is crucial for skin health and tumor development.

FOXN1 duplication can cause excessive hair growth.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Bioengineered lacrimal glands can restore tear production and protect eyes.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.

Y27632 increases cell growth through EGFR signaling, not ROCK1/2.

Phospholipids help plant proteins move by regulating receptor interactions.

Lef1 is essential for normal skin, hair growth, and healing wounds in mice.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Certain genes controlled by OVOL1 are crucial for creating new hair follicles.

A genetic variant in the GJB6 gene likely caused the Labrador's paw pad condition.

Glucosylceramides are essential for healthy skin and proper wound healing.

PINCH-1 is crucial for skin cell adhesion and movement, working with EPLIN and ILK.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

HEY2+ cells help regenerate skin during wound healing.

A gene mutation in mice causes permanent hair loss and skin issues.