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Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Mutations in the DSG4 gene cause a severe form of brittle hair and skin issues.

HAT-L4 is crucial for preventing body fluid loss by maintaining skin barrier integrity.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Tribuloside can increase skin pigmentation by enhancing melanin production and distribution.

GLABRA2 gene controls root-hair growth by regulating phospholipid signaling.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

A new genetic mutation linked to Hutchinson-Gilford progeria syndrome was found in China.

Agalsidase alfa helps treat Fabry's disease but needs more research for long-term benefits.

The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Certain astrocytes can protect the brain and improve recovery after a stroke, and a hair loss drug might reduce cancer spread.

ErbB2 signaling is crucial for skin cell growth and cancer development in mice.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

A rare skin tumor with extra hair growth was found and safely removed from a 27-year-old woman.

TBX5 gene influences feathered feet in Guangxi chickens by affecting cell growth and movement.

High levels of human keratin 16 in mice cause skin lesions and abnormal skin development.

The peptide IMT-P8 can effectively deliver proteins into the skin and cells for potential skin treatments.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

The muscle damage was caused by T-cell large granular lymphocytic leukemia, not dermatomyositis.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

A patient with a new PLEC gene mutation showed symptoms of both muscular dystrophy and myasthenia gravis, which improved with steroid treatment.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

MOF controls skin development by regulating genes for mitochondria and cilia.

Aδ-LTMRs have complex synapses with glycine, while Aβ-LTMRs have simpler ones.