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Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

The rhg mutation in mice affects the Oat gene, causing hair growth issues and other symptoms.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

RXRα is crucial for proper immune response and links diet to immune function.

A gene mutation in mice causes skin defects and early death.

Removing hair from mice without reproductive glands led to grey hair, possibly helping to understand greying in aging.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

Mice with human gene experienced hair loss when treated with DHT.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Suppressing the HGPS mutation may improve symptoms and suggest reversibility.

Gamma-ray exposure improves genome editing efficiency in mice using the i-GONAD method.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

The G(S) alpha subunit gene may help start hair follicle growth in newborn mice.

The mutation helps mice handle heat better without affecting hair growth.

The N gene affects the protein makeup of mouse hair.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

Vitamin D receptor is crucial for starting hair growth after birth.

GFP transgenic mice help study cell origins in skin grafts.

Using special RNA to target a mutant gene fixed hair problems in mice.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Mouse models help study genetic skin diseases.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.