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Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Pannexin 3 helps skin and hair growth by controlling a protein called Epiprofin.

A protein called HIF-1a helps control hair growth genes and could be targeted to treat hair loss.

ETS2 drives cancer progression in squamous cell carcinoma and is linked to poor patient outcomes.

Ancient DNA blocks are still present in human genomes, possibly due to advantages they provide.

Protein Kinase Cε increases skin sensitivity to UV damage and skin cancer risk.

Breast cancer alters specific molecular structures in hair, which revert after cancer removal.

Polyamines help fix DNA damage accurately in cells.

KRT17 may be a new target for endometrial cancer treatment because it helps cancer cells move and form new blood vessels.

Histamine and its enzyme may help start hair regrowth.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

Small amounts of DNA can be found in the hair shaft, especially near the root, but it decreases with hair treatments and washing.

A specific genetic marker is linked to male pattern baldness in Han Chinese men.

NuMA-microtubule interactions are vital for proper skin structure formation and function.

DNA methylation and long non-coding RNAs are key in controlling hair growth in Cashmere goats.

A gene mutation causes curly hair and hair loss in rats.

Hairless mammals evolved quickly in both gene and non-gene areas related to skin and hair.

Different parts of the nail express different keratins, showing unique patterns of differentiation.

MCHR2 gene duplications may be linked to alopecia areata.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

Spermidine may improve skin health and hair growth by enhancing cell function.

TRPS1 loss in balding scalp areas contributes to hair loss in androgenic alopecia.

The ventral matrix is the main source of the nail plate.

Researchers made a detailed map of gene activity for different parts of human hair follicles to help create targeted hair disorder treatments.

Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.