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The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

A genetic mutation in the hHa1 gene creates a smaller, but still functional, hair protein without causing hair problems.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Scientists discovered two versions of a new human hair keratin gene.

A protein called GIS3 is important for the growth of root hairs in Arabidopsis by controlling two genes with the help of certain growth signals.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Increased Stat3 activity reduces hair follicle stem cells and boosts other stem/progenitor cells.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

Myc changes chromatin in stem cells, causing them to leave their niche.

The study found potential new DNA patterns in fertility genes, but further testing is needed.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

TCDD changes skin cell growth and keratin production in mice.

Estrogen and MLL enzymes work together to regulate genes important for hair growth and leukemia.

The Itpr3 gene causes a specific hair pattern in mice.

The DMI3 gene is essential for nodule development and symbiosis in certain plants.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

HDAC4 and HDAC7 are crucial for Th17 cell development and could be targeted to treat inflammatory diseases.

A second domain of high sulfur KAP genes on chromosome 21q23 is crucial for hair structure.

DNA methylation of miR-365-1 causes hair follicle stem cell death by affecting DAP3.

N6-methyladenosine affects hair follicle development differently in Rex and Hycole rabbits.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

DNMT1 helps turn hair follicle stem cells into fat cells by blocking a specific microRNA.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Mouse hair keratins mHa1 and mHb4 can't form a strong network on their own in cells.