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LGR5 is a common marker of hair follicle stem cells in different animals and is important for hair growth and regeneration.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

The 14-3-3σ gene is essential for preventing hair loss.

Researchers found new genes involved in hair growth, which could help develop new hair treatments.

Slug (Snai2) helps regulate hair growth timing in mice.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

Researchers found 71 genetic regions linked to male pattern baldness, which account for 38% of its genetic risk.

A mutation in the KRT74 gene causes tightly curled hair.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

A specific genetic marker is linked to male pattern baldness in Han Chinese men.

Four genetic risk areas related to male-pattern baldness were identified, with WNT signaling playing a role in its development.

Four genetic risk spots found for hair loss, with WNT signaling involved and a link to curly hair.

Hypertrichosis lanuginosa causes excessive fine hair growth, often linked to genetics or cancer, with limited treatment options.

Grafted human scalp samples on mice can produce human hair, useful for studying hair genetics.

The gene for slick hair in Senepol cattle is located on chromosome 20 and may involve the SRD5A2 gene.

A new gene mutation is linked to monilethrix in the studied family.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

Some cases of short anagen hair syndrome are linked to specific genetic variations that are also connected to male pattern baldness.

The study maps how genes are regulated during mouse hair growth.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

Wnt ligands, produced by dermal papilla cells, are essential for adult hair growth and regeneration.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

The gene HDC is important for the development of hair follicles in newborn mice.

A new mutation in the HR gene is linked to a rare form of hair loss with limb deformities.